Autoimmunity potentially benefits from the extended exposure to nuclear and intracellular autoantigens derived from late apoptotic and secondary necrotic cells. B and T cell tolerance for apoptotic cells is abrogated, as well as lymphocytes get activated by these autoantigens; inflammation and also the manufacture of autoantibodies by plasma cells is https://emilio186z7.look4blog.com/54101157/detailed-notes-on-who-discovered-allergies